Supplementary MaterialsSupplemental Desk 1
Human Neutrophil Elastase 0 CommentsSupplementary MaterialsSupplemental Desk 1. appearance of proliferative, homeostatic and effector genes. The neuropeptide receptor was expressed by ILC2s at steady state and after IL-25 stimulation preferentially. Neuromedin U (NMU), the ligand of NMUR1, turned on ILC2s co-administration of NMU with IL-25 amplified allergic inflammation strongly. Lack of NMUCNMUR1 DB07268 signalling decreased ILC2 effector and regularity