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Many ovarian malignancies originate from the ovarian surface area epithelium and are characterized simply by aneuploid karyotypes. diploid cells, and gave rise to aneuploid cells through chromosome mis-segregation during both multipolar and bipolar mitoses. Shot of the past due passing aneuploid MOSECs lead in growth Acadesine development in C57BM/6 rodents. As a result, we reveal

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