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BACKGROUND Supplement D attenuates uremic cardiac hypertrophy, possibly by suppressing the myocardial reninCangiotensin program (RAS) and fibroblast development factors (FGFs). neglected group was likewise attenuated by Computer or E. Upregulation of myocardial expressions of renin, angiotensinogen, FGFR-1, and BNP in neglected uremic pets was decreased similarly by Computer and E, as the angiotensin II type

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